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"IGF-I triggers both hypertrophy and hyperplasia through stimulation of satellite cell proliferation and differentiation, particularly with autocrine/paracrine signaling." — Adams GR, Journal of Applied Physiology (2002) Practically, this is the mechanistic rationale behind site-specific DES injection into a lagging muscle belly: localized saturation of satellite-cell IGF-1R, at the moment of training-induced damage, is where the hyperplasia signal is strongest

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Regulation of tumor initiation by the mitochondrial pyruvate carrier