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Oxidative stress as a potential modulator of hormonal signaling OS may modulate the biological effects of E₂ and progesterone through multiple pathways, with its primary mechanisms involving two aspects: synergistic effects with hormone signaling in key pathways, and potential influence on hormone receptor expression and function

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120 In myeloid cell-specific SIRT1 knockout mice, lipid toxicity or LPS stimulation induced more severe hepatic inflammation, elevated pro-inflammatory cytokine levels, and enhanced M1 macrophage polarization, highlighting the critical role of SIRT1 in preserving immune balance